If you were to ask a group of people why brain cancer would rise to the surface, the various answers given will show that there is no concrete reasoning. Enzymes could play a role in the matter, as it's been said that they can trigger and make this condition progress. This is especially true when focusing on glioblastoma, the most common and aggressive type of cancer in this regard. Focusing on enzymes, though, what can be done in order to stop them in their tracks?
Futurity posted an article that went into detail about a method that could come into play. It was described as a "backdoor" approach that would be followed through with in order to help brain cancer. The ability to halt an enzyme related to glioblastoma is one that deserves to be considered, as organizations such as Voices against Brain Cancer would be able to tell you. The details on the matter may not be made clear, though, so what exactly do these include, one has to wonder?
The enzyme that I would like to focus on in regards to this study goes by the name of phospholipase D. While it is one that has been connected to brain cancer, it is prudent to make note of how it is connected to other forms of cancer, gastric being just one instance to consider. PLD also comes into play for the sake of regulating Akt. For those who are not in the know, Akt is another enzyme that specifically plays into the growth and progression of cancer.
PLD was given focus in this regard and it was done during a study that involved afflicted mice. Judging by the information previously stated, one would imagine that blocking the actions of Akt would prove to be of great assistance to the issue here. However, the article said that if it is blocked, the body stands a great chance of triggering an exaggerated immune response or diabetic symptoms. Akt has to be targeted, of course, but there should still be a level of urgency taken into account.
According to Professor Craig Lindsley, as the article stated, it is possible to modulate the one isoform associated with Akt without bringing harm to the patient. Without Akt being activated in the body, the cancerous cells in the body die, which does not negatively impact the patient in question in any way. This is especially vital in this field of research. As long as the implications do not long-lasting results, I do not think that anyone can argue with the level of potential seen here.
Futurity posted an article that went into detail about a method that could come into play. It was described as a "backdoor" approach that would be followed through with in order to help brain cancer. The ability to halt an enzyme related to glioblastoma is one that deserves to be considered, as organizations such as Voices against Brain Cancer would be able to tell you. The details on the matter may not be made clear, though, so what exactly do these include, one has to wonder?
The enzyme that I would like to focus on in regards to this study goes by the name of phospholipase D. While it is one that has been connected to brain cancer, it is prudent to make note of how it is connected to other forms of cancer, gastric being just one instance to consider. PLD also comes into play for the sake of regulating Akt. For those who are not in the know, Akt is another enzyme that specifically plays into the growth and progression of cancer.
PLD was given focus in this regard and it was done during a study that involved afflicted mice. Judging by the information previously stated, one would imagine that blocking the actions of Akt would prove to be of great assistance to the issue here. However, the article said that if it is blocked, the body stands a great chance of triggering an exaggerated immune response or diabetic symptoms. Akt has to be targeted, of course, but there should still be a level of urgency taken into account.
According to Professor Craig Lindsley, as the article stated, it is possible to modulate the one isoform associated with Akt without bringing harm to the patient. Without Akt being activated in the body, the cancerous cells in the body die, which does not negatively impact the patient in question in any way. This is especially vital in this field of research. As long as the implications do not long-lasting results, I do not think that anyone can argue with the level of potential seen here.
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